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Content archived on 2024-05-27

Investigating epigenetic silencing mechanisms within cancer

Objective

Epigenetic silencing is essential for modulating gene expression patterns in developing organisms and has very big impact on the genome integrity. Cancer, as well as many other diseases, has been linked to the changes in transcription of regulatory genes due to the aberrant epigenetic silencing. Despite many epigenetic regulators, enzymes including histone deacetylases, methyltransferases, and DNA methyltransferases, were discovered, it is critical to bridge the gap in our understanding of the mechanisms contributing to chromatin structure modulation. We employed a phenotypic screening assay in mouse erythroid leukemia cells that harbour a silenced green fluorescent protein (GFP) reporter transgene in order to isolate new components of heterochromatic silencing and identify compounds that have potential to de-repress tumour suppressor genes in cancer cells. Strikingly among the compounds with strong anti-silencing properties we found inhibitors of bromo and extra-terminal domain (BET) proteins, which recently became massively popular as new generation chemotherapy anti-cancer drugs. Since so far BET proteins were mainly considered to act as transcriptional co-activators, we have set out to investigate the molecular mechanism of action of the BET inhibitors with respect to their anti-silencing properties. Our preliminary data suggests that inhibition of BET proteins results in loss of the DNA methylation, and specific histone modifications from heterochromatic regions. The aims of this proposal are: 1) To determine which BET proteins are important for heterochromatin maintenance; 2) To determine the mechanism of action of the key BET protein(s) in epigenetic silencing; 3) To analyse chromatin changes resulting from inhibiting of the key BET protein(s) in the selected cancer cell lines. We expect that this research will not only shed light on fundamental principles of genome organization, but also will instruct cancer research and lead to new therapeutic approaches.

Fields of science (EuroSciVoc)

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Topic(s)

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Call for proposal

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FP7-PEOPLE-2013-IEF
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Funding Scheme

Funding scheme (or “Type of Action”) inside a programme with common features. It specifies: the scope of what is funded; the reimbursement rate; specific evaluation criteria to qualify for funding; and the use of simplified forms of costs like lump sums.

MC-IEF - Intra-European Fellowships (IEF)

Coordinator

THE UNIVERSITY OF EDINBURGH
EU contribution
€ 221 606,40
Address
OLD COLLEGE, SOUTH BRIDGE
EH8 9YL Edinburgh
United Kingdom

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Region
Scotland Eastern Scotland Edinburgh
Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

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