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Understanding how long non-coding RNAs adapt the adipocyte for specialised lipid storage and breakdown.

Project description

Non-coding RNAs and the metabolic processes of adipocytes

Adipocytes (fat cells) store energy as lipids, which can be broken down into fatty acids and released into the circulation when needed. Under normal circumstances, correct storage and release of lipid maintains metabolic health. However, in the case of obesity, the balance of lipid storage and release dysfunctions which can lead to insulin resistance, diabetes and other cardiometabolic diseases. The ERC-funded LNC-LIPID project aims to investigate how long non-coding RNAs (lncRNAs) promote adipocyte lipid storage and release. Using innovative methodologies to identify lncRNA-interacting proteins, it will pinpoint disease-regulated lncRNAs in adipose tissue biopsies and test how they function in human cells. The findings will offer insight into how lncRNAs affect lipid metabolism and contribute to metabolic health.

Objective

Energy is stored in adipocytes as a large uniocular lipid droplet, which can be broken down into fatty acids (FAs) and released into the circulation when needed. This specialised lipid storage and breakdown requires dynamic organisation of metabolic enzymes and cofactors around the large lipid droplet for metabolic homeostasis. In obesity, the adipocyte dysfunctions, resulting in high spontaneous release of FAs. This chronic release and elevation of systemic FAs is a major driver of systemic insulin resistance, type 2 diabetes and other cardiometabolic diseases.

I postulate that long non-coding RNAs (lncRNAs) interact with the metabolic machinery within the adipocyte to organize cell- and stimulation-specific interactomes, channelling substrate flux within the cell. Adipocytes express over 4000 lncRNAs, many of which are unique to humans and this cell type. However, whether these lncRNAs contribute to the adipocyte’s specialised lipid metabolism in health and disease is not understood. This proposed research aims to address this question.

I have already developed key methodologies to study lncRNAs in human adipocytes, including a novel technique called TROOPS that identifies specific lncRNA-interacting proteins. Leveraging an extensive bank of white adipose tissue biopsies from uniquely characterized patients, I will identify disease-regulated lncRNAs and test how they function in human adipocytes. I will use gene editing techniques and lipid/metabolomics analysis to define the role of these lncRNAs in lipid storage and FA release.

Furthermore, I will use protein-lncRNA complex purification combined with advanced microscopy techniques to reveal how lncRNAs can sequester protein complexes into phase-separated organelles and organize adipocyte lipid metabolism. These insights will provide a paradigm shift in understanding how lncRNAs enable the adipocyte to perform specialized functions and new generalizable findings for how lncRNAs contribute to cell function.

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HORIZON-ERC - HORIZON ERC Grants

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(opens in new window) ERC-2024-STG

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Host institution

KAROLINSKA INSTITUTET
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 493 925,00
Address
NOBELS VAG 5
171 77 STOCKHOLM
Sweden

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Region
Östra Sverige Stockholm Stockholms län
Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 493 925,00

Beneficiaries (1)

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