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Deciphering translocation-based genome topology effects and their role in lymphoma formation

Project description

Chromosomal translocations and nuclear function

Cancer is often associated with chromosomal translocations, a phenomenon whereby a chromosome breaks, and its fragments reattach to another chromosome. This may interrupt tumour-suppressor genes or more often create fusion genes or proto-oncogenes at the translocation breakpoint, leading to oncogenesis. The working hypothesis of the EU-funded LymphoTOP project is that chromosomal translocations disturb genome architecture and may cause further deregulation. Researchers will focus on non-Hodgkin lymphoma and determine how translocations in immunoglobulin genes may affect the genomic and epigenetic landscape and lead to the disease. Results will provide fundamental knowledge on nuclear function and carcinogenesis.

Objective

A central goal in cancer research is to identify the molecular mechanisms underlying tumor formation, such as translocations that result in expression of fusion genes or proto-oncogenes located at the translocation breakpoints. While the consequences of this erroneous expression have been widely studied, other effects of translocations remain largely unexplored. Nevertheless, these can provide vital new insights into tumorigenesis. I hypothesize that translocations disturb the global 3D genome architecture, causing alterations in chromosome territory positioning and interchromosomal interactions. And, that these can account for (in)activation of key genomic loci for which the cause of deregulation remains so far unknown. I will address this hypothesis in the context of Non-Hodgkin lymphoma (NHL)-related immunoglobulin (IGH) translocations. By genome editing we will create lymphoblastoid cell lines and cord blood cell-derived B lymphocytes in which IGH translocation-based effects can be cleanly dissected in vitro and in vivo. In these models, we will study how IGH translocations affect genome topology, the epigenetic and gene expression landscape and NHL formation. To that end, we will assess (i) chromosome territory structure and interchromosomal crosstalk by chromatin conformation capture and fixed- and live-cell imaging, (ii) epigenetic and gene expression maps using bulk and single-cell profiling and (iii) in vivo tumor formation. Importantly, after exploring the presence of the observed gene expression changes in NHL samples, we will target the coinciding changes in in vitro and in vivo approaches to probe their relevance for lymphomagenesis. Overall, LymphoTOP shall lead to deeper insights into translocation-based genome topology reorganisation and its role in tumorigenesis, and to potential identification of new vulnerabilities of lymphomas. Thus, extending its impact to many fields, from lymphoma and tumor biology to 3D genome organisation and beyond.

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HORIZON-ERC - HORIZON ERC Grants

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Call for proposal

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(opens in new window) ERC-2021-STG

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Host institution

FUNDACIO CENTRE DE REGULACIO GENOMICA
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 499 993,00
Address
CARRER DOCTOR AIGUADER 88
08003 Barcelona
Spain

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Region
Este Cataluña Barcelona
Activity type
Research Organisations
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 499 993,00

Beneficiaries (1)

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