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The role of GPCRs and homing molecules in the control and regionalization of mucosal immunity.

Project description

Molecular mechanisms of mucosal immunity

Mucosal surfaces throughout the body, such as in the linings of the gastrointestinal and respiratory tracts are the primary points of contact with the external environment. Mucosal immunity plays a central role in protecting against infection but also preventing inflammatory responses. Funded by the European Research Council, the GREMLIN project is interested in delineating the molecular pathways that govern mucosal-associated lymphoid tissues (MALTs), specialised immune structures in mucosal surfaces. Researchers will explore the role of G protein-coupled receptors (GPCRs) on the recruitment and positioning of immune cells within MALTs during inflammation. Project findings have the potential to determine GPCRs as therapeutic targets for clinical interventions.

Objective

Mucosal immunity is essential to preserve homeostasis and for pathogen control within the gut and the airways. The emerging dogma of immune regionalization has opened to new questions about which elements drive cell adaptation to different niches, and how this can be targeted to cure diseases. The intestine and the airways are functionally diverse along their length, and disorders can differentially impact on different mucosal tissue regions. Mucosa-associated lymphoid tissues (MALTs) are located along the intestine and the airways to drain selective tissue segments and support protective immunity. Whether MALTs located in different tissue areas show immune regionalization remains poorly understood. Also, little is known about which molecules control such immune diversity. G protein-coupled receptors (GPCRs) are surface proteins important for cell migration and compartmentalization in lymphoid tissues, but their role in immune regionalization is not clear. We propose to apply a combination of imaging, flow cytometry, spatial gene editing and sequencing techniques to study the role of GPCRs in immune regionalization of MALTs, at steady state and in disease. In the intestine, we will phenotypically and functionally profile immune cells and GPCRs expression within Peyer's patches (PPs) from different gut regions, in physiology or in the context of inflammation and obesity. Similarly, we will analyze immune regionalization of bronchus-associated lymphoid tissues and nose-associated lymphoid tissues within the airways, at steady state or upon infections. Finally, we will characterize the role of a candidate GPCR, GPR35, in the induction of intestinal and respiratory immunity. Our preliminary data show that GPR35 shapes cell recruitment to inflamed airways and influences cell positioning within PPs. By applying the above-mentioned approaches and dedicated mouse models, we will study how GPR35 regulates mucosal immunity and if it represents an attractive clinical target.

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Topic(s)

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HORIZON-ERC - HORIZON ERC Grants

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Call for proposal

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(opens in new window) ERC-2023-STG

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Host institution

UNIVERSITA VITA-SALUTE SAN RAFFAELE
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 500 000,00
Address
VIA OLGETTINA 58
20132 Milano
Italy

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Region
Nord-Ovest Lombardia Milano
Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 500 000,00

Beneficiaries (1)

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