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Targeting eicosanoid metabolism to overcome tumor immunosuppression

Project description

Targeting the tumour microenvironment for enhanced immunotherapy

The tumour microenvironment (TME) is a complex network of cells embedded in an altered extracellular matrix that surrounds and supports tumours. It plays a crucial role in cancer progression, often fostering an immunosuppressive environment that helps tumours evade immune detection and treatment. The ERC-funded EicoMet project aims to target tumour-associated macrophages (TAMs), one of the most abundant and immune-suppressive cell populations in the TME. To reprogramme TAMs from an immunosuppressive state, researchers will inhibit the enzyme HPGDS. This metabolic editing is expected to enhance immune cell infiltration in the tumour and overcome resistance to current treatments, offering new opportunities for combination therapies.

Objective

The field of cancer immunotherapy has witnessed exciting therapeutic advances in the last decade, but still a large fraction of patients is not responsive and several cancer types are refractory to this therapy. The presence of a strongly immunosuppressive environment is a likely explanation for the suboptimal efficacy of immunotherapy. One of the most abundant and immune suppressive population in the tumor microenvironment (TME) is represented by tumor-associated macrophages (TAMs). Therefore, several strategies that sculpt the TAM phenotype away from their immunosuppressive state are expected to become the next generation immunotherapies.
Supported by the ERC consolidator grant ImmunoFit, we recently unveiled that the metabolic editing of the TME via the pharmacological and genetic inhibition of HPGDS in macrophages (an enzyme involved in the production of prostaglandin D2 from arachidonic acid), represents a novel therapeutic strategy to sculpt TAMs away from their immunosuppressive state, increase infiltration and fitness of CD8+ T cells and NK cells, and promote tumor vessel normalization. This change in the TME upon HPGDS inhibition overcomes resistance to current immunotherapeutic regimens in melanoma and other cancer indications. This proposal aims to de-risk HPGDS targeting by delivering a lead compound with proven efficacy in several preclinical models. The deliverables of this project will open-up a window of opportunities in immune-oncology and lead to novel combination therapies by increasing the potency of the current therapeutic approaches.

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HORIZON-ERC-POC - HORIZON ERC Proof of Concept Grants

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Call for proposal

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(opens in new window) ERC-2023-POC

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Host institution

VIB VZW
Net EU contribution

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€ 150 000,00
Address
SUZANNE TASSIERSTRAAT 1
9052 ZWIJNAARDE - GENT
Belgium

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Region
Vlaams Gewest Prov. Oost-Vlaanderen Arr. Gent
Activity type
Research Organisations
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Total cost

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