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The unusual role of a highly divergent Arp2/3 complex in the mosquito stages of malaria parasites.

Project description

Understanding Plasmodium parasites for antimalarial strategies

Plasmodium parasites, the cause of malaria, rely on unique proteins for sexual replication in mosquitoes, yet their development is not fully understood. A recently discovered actin-related protein 2/3 (Arp2/3) complex plays a crucial role in DNA segregation and oocyst development, but its activation mechanisms are still unclear. The ERC-funded PlasmoArp project will investigate the activation of the Plasmodium Arp2/3 complex during gamete formation and its unique regulatory mechanisms. It will explore the connection between Arp2/3 and a previously unknown spindle assembly checkpoint regulating mitosis. The project will also examine why Arp2/3-deficient parasites arrest in oocysts, shedding light on the cell division processes. PlasmoArp will make use of molecular biology techniques, single-cell transcriptomics and advanced imaging and develop a new genetic tool for dissecting oocyst-specific gene function.

Objective

The malaria-causing Plasmodium parasites often use highly divergent proteins to regulate fundamental biological processes, such as sexual replication in the mosquito. Therefore, the molecular and cellular biology underlying parasite development is often little understood, especially during the mosquito stages, a major bottleneck in the Plasmodium life cycle. Recently, I identified an unconventional, Plasmodium-specific actin-related protein 2/3 (Arp2/3) complex that mediates DNA segregation during male gamete formation and is essential for development of the main replicative mosquito stage, the oocyst. Understanding the regulation and function of this structurally and functionally divergent actin nucleator could provide targets for new antimalarial strategies. Here, I propose to elucidate the molecular mechanism of the Plasmodium Arp2/3 complex on three levels: (1) I will uncover how Arp2/3 is activated during gamete formation, revealing the likely unusual mode of regulation of this complex. (2) I will explore the hypothesis that Arp2/3 is linked to a cryptic spindle assembly checkpoint, a mitosis regulator so far thought to be absent in Plasmodium. (3) I will reveal why Arp2/3-deficient parasites arrest in oocysts, which will shed light on the unusual cell division mode of this elusive stage. To address these ambitious aims, I will combine molecular biology methods, single cell transcriptomics and imaging, and I will engineer a new genetic tool to dissect gene function specifically in the oocyst, a stage that is notoriously difficult to study. PlasmoArp will thus not only give insight into the molecular biology of Plasmodium development in the mosquito and advance our understanding of Arp2/3 and actin biology across the eukaryotic kingdom, but it will also expand the toolbox to study the neglected oocyst. As Plasmodium Arp2/3 is essential for malaria transmission, this research will pave the way for new intervention strategies to mitigate malaria infections.

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(opens in new window) ERC-2024-STG

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Host institution

UNIVERSITATSKLINIKUM HEIDELBERG
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 499 527,00
Address
IM NEUENHEIMER FELD 672
69120 HEIDELBERG
Germany

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Region
Baden-Württemberg Karlsruhe Heidelberg, Stadtkreis
Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 499 527,00

Beneficiaries (1)

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