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Targeting the Plant Cysteine Oxidases to Regulate Plant Stress Tolerance

Project description

Developing tools to improve plant stress tolerance

Crop loss due to weather extremes affects the world’s poorest communities and also has widespread global impact. Tools and strategies such as the molecular engineering of crops are therefore needed to improve crop tolerance to such abiotic stress. Group VII ethylene response transcription factors (ERF-VIIs) play an important role in plant stress tolerance. Although ERF-VIIs are destabilised when their N-terminal cysteine residues are oxidised, plant cysteine oxidase enzymes (PCOs) control ERF-VII stability and mediate the response to flood-induced hypoxia. The EU-funded PCOMOD project aims to generate tools to manipulate PCO activity, modulate cys-sulfinic acid formation and stabilise ERF-VIIs. This work will help create effective and targeted mechanisms to manipulate PCO activity and improve stress tolerance in crops.

Objective

Population growth and climate change mean that food security is an emerging global challenge. Crop loss due to flood, drought and other weather extremes is something that disproportionately affects the world's poor, but also has widespread international impact. There is an immediate and urgent need to develop tools and strategies to improve crop tolerance to such abiotic stress.

One effective mechanism towards this goal is molecular engineering of crops to withstand prolonged abiotic stress. Group VII Ethylene Response transcription Factors (ERF-VIIs) have a key role in plant stress tolerance, in particular flooding but also salinity, high temperature, drought and oxidative stress. ERF-VIIs are readily degraded, but their stabilisation has led to improved flood tolerance in model plants and crops. Consequently, ERF-VIIs are focal points for engineering abiotic stress resistance in crops.

ERF-VIIs are destabilised when their N-terminal cysteine (Nt-Cys) residues are oxidised, making them substrates for the N-end rule pathway of protein degradation. I discovered that Plant Cysteine Oxidase enzymes (PCOs) catalyse this oxidation, incorporating molecular oxygen into ERF-VII Nt-Cys residues to form Cys-sulfinic acid (CSA), and that PCO activity is sensitive to oxygen availability. These enzymes therefore control ERF-VII stability and mediate the response to flood-induced hypoxia.

I propose generating tools and knowledge to manipulate PCO activity, modulate CSA formation and stabilise ERF-VIIs. This is an attractive and tractable strategy to enhance stress tolerance in plants. The project will require (i) the development of efficient tools and assays to detect and quantify CSA, (ii) an understanding of the breadth of PCO activity for non-ERF-VII substrates, and (iii) an understanding of the role of non-enzymatic CSA formation. This knowledge will enable the development of effective and targeted mechanisms to manipulate PCO activity and improve stress tolerance.

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Programme(s)

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Topic(s)

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Funding Scheme

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ERC-COG - Consolidator Grant

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Call for proposal

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(opens in new window) ERC-2019-COG

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Host institution

THE CHANCELLOR, MASTERS AND SCHOLARS OF THE UNIVERSITY OF OXFORD
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 995 253,00
Address
WELLINGTON SQUARE UNIVERSITY OFFICES
OX1 2JD Oxford
United Kingdom

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Region
South East (England) Berkshire, Buckinghamshire and Oxfordshire Oxfordshire
Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 995 253,00

Beneficiaries (1)

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