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The biology of syncytial cells: Dissecting the mechanisms and functions of nuclear differentiation inside skeletal muscle syncytium

Project description

Insight into the nuclear diversity of muscle

Muscle fibres are an example of syncytial cells where individual muscle cells are joined together to improve structural stability and facilitate the contraction of the entire fibre. Funded by the European Research Council, the SyncyNucDiff project will address a fundamental question in syncytia biology: how do these cells organise gene expression among the different nuclei? Previous work has indicated great diversity in the transcriptional programme of each myofibre nucleus. To understand how these identities are maintained, researchers will focus on the role of chromatin architecture and transcriptional regulators. Project results and the innovative tools created will pave the way for studying the pathophysiology of syncytia cells in the near future.

Objective

Due to their cellular anatomy, syncytial cells face many unique challenges that mono-nucleated cells do not. One of them is how syncytial cells organize gene expression among many nuclei in a shared cytoplasm. How such property is linked to their biological functions remains poorly understood. Using the skeletal muscle as a paradigm and single-nucleus transcriptomics, I recently uncovered previously unrecognized diversity and dynamics of myonuclear transcriptional programs. This conceptualized the syncytial muscle cell itself as an analog of multi-cellular tissue where individual nuclei are counterparts of differentiated cell types. My findings raise two important and unanswered questions. 1) How are the diverse nuclear identities specified and maintained? And 2) What are the functional contributions of the diverse nuclear subtypes in health and disease? I will first focus on the nuclei at the neuromuscular junction (NMJ) and myotendinous junction (MTJ), which are responsible for initiation of contraction and dissipation of contractile force, respectively. Despite their well-established functions, the mechanisms that specify or maintain them are sparsely understood. I will investigate how chromatin architecture and transcriptional regulators govern their identities. I will then characterize the upstream signalling pathways from motor neuron or tendon cells that activate the specific transcriptional programs. So far, genetic manipulation of myofibers have targeted entire muscle nuclei. To overcome this limit, I will develop tools that allow genetic manipulation in specific nuclear subtypes. Previous works had identified a new nuclear subtype associated with muscular dystrophy and a transient time window during postnatal development. I will characterize the function of these nuclei using a method that abolishes their transcriptional activities. The approaches to be developed here will pave the way to understanding the pathophysiology of syncytial cells in the future.

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Call for proposal

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(opens in new window) ERC-2021-STG

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Host institution

INSTITUT NATIONAL DE LA SANTE ET DE LA RECHERCHE MEDICALE
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 055 625,00
Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 055 625,00

Beneficiaries (2)

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