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Development of artificial phosphoinositides aiming at HIV eradication

Descrizione del progetto

Strategia «Lock-in e apoptosi» per combattere l’HIV

Il virus dell’immunodeficienza umana (HIV), che finora ha mietuto più di 40 milioni di vittime, continua ad essere un grave problema di salute pubblica. Nonostante gli sforzi per affrontare l’epidemia di HIV/AIDS, non si è riusciti del tutto nell’intento. Recenti osservazioni indicano che, anche dopo il trattamento, nell’organismo rimane un serbatoio virale estremamente resistente alla terapia farmacologica antiretrovirale. Il progetto Super-HIPPO, finanziato dal programma di azioni Marie Skłodowska-Curie, svilupperà una nuova strategia chiamata «lock-in e apoptosi». La strategia si basa su un derivato non naturale dell’esafosfato di inositolo, L-HIPPO, che nel corso della sperimentazione ha mostrato un forte effetto sulle cellule HeLa infettate dall’HIV. Ora l’obiettivo è sviluppare un nuovo derivato della L-HIPPO, più efficiente, che possa essere somministrato per via orale.

Obiettivo

Main challenge of fighting with the HIV is to eliminate the latent viral reservoir from the body. This reservoir is resistant to antiretroviral drug therapy and can cause viral rebound if the treatment is stopped. The removal of the reservoir enables HIV eradication, is urgently desired in HIV-AIDS research. For this purpose, a strategy called “kick and kill” was proposed, based on the hypothesis that activation of latent HIV (“kick”) leads to cell death (“kill”) by physical damage and/or immune activation. However, in clinical tests “kill” process was found to be not enough to reach HIV eradication.
To eradicate HIV from the body, my work has recently suggested a new strategy called “lock-in and apoptosis” instead of “kick and kill”. In development of this strategy, non-natural derivative of inositol hexaphosphate (IP6) named as L-HIPPO was designed based on the fact that the MA domain of Pr55gag which mediates membrane binding through its interaction with inositol phospholipid PIP2 in the host membrane. L-HIPPO was administrated to HIV infected HeLa cells as complex with a carrier α-CDE and suppressed membrane localization of HIV-1 Gag protein and induced strong apoptosis of the host cell containing the latent viruses. In contrast, α-CDE-L-HIPPO induced less apoptosis on T cell line. Besides, the complex of α-CDE-L-HIPPO is inapplicable for oral use. Toward this end, new L-HIPPO derivative with alternative carrier is required.
An ultimate goal of this work is to develop new L-HIPPO derivative (Super-HIPPO) with more potent and efficient activities. My objectives to reach this goal are (1) to synthesize new L-HIPPO derivatives and alternative carriers, (2) to evaluate their activities, (3) to repeat design, synthesis and biological evaluation, if the activities are not enough, (4) to confirm the binding mode of MA-compound, and (5) to achieve personal development and career advancement by performing this study.

Campo scientifico (EuroSciVoc)

CORDIS classifica i progetti con EuroSciVoc, una tassonomia multilingue dei campi scientifici, attraverso un processo semi-automatico basato su tecniche NLP.

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Coordinatore

IZMIR KATIP CELEBI UNIVERSITESI
Contribution nette de l'UE
€ 148 478,40
Indirizzo
IZMIR KATIP CELEBI UNIVERSITESI HAVAALANI SOSESI C NO.33, D.2
35620 İzmir
Turchia

Mostra sulla mappa

Regione
Ege İzmir İzmir
Tipo di attività
Higher or Secondary Education Establishments
Collegamenti
Costo totale
Nessun dato