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Shaping the bacterial envelope: Interplay between the components and impact on antibiotic resistance

Project description

Bacterial envelope interactions impacting antibiotic resistance

Gram-negative bacteria are resilient pathogens, primarily due to their tripartite envelope, which consists of the inner membrane, peptidoglycan cell wall, and outer membrane. Recent studies highlighted a crucial interaction between PG and the BAM complex, which coordinates the insertion of outer membrane proteins (OMPs), a process essential for proper envelope assembly. The ERC-funded Shape-En-Resist project will study the interactions that influence bacterial physiology and antibiotic resistance. It focuses on the assembly of lipopolysaccharide (LPS), its relationship with envelope biogenesis, and how these affect bacterial responses to antibiotics. The project also investigates the spatially heterogeneous nature of envelope biogenesis as a source of phenotypic diversity. It will combine microscopy, genetic screening, proteomics, and functionality assays.

Objective

Gram-negative bacteria are notoriously resilient, dominating the list of pathogens requiring urgent medical attention. Fundamental to their remarkable antibiotic resistance is a tripartite envelope comprised of an inner-membrane (IM), peptidoglycan cell wall (PG) and an outer membrane (OM). While these layers were previously viewed as separate compartments, it is becoming apparent that they work together in order to maintain envelope integrity. However, little is known about the crosstalk between components and molecular mechanisms orchestrating envelope assembly and functionality.
Our latest work has identified a key interaction between the PG and the BAM complex which inserts new OM proteins (OMPs), revealing a mechanism coordinating PG and OMP biogenesis and directing them both to the cell center. We posit that coordination is a key principle of envelope assembly and multiple interactions among envelope constituents are waiting to be discovered. In Shape-En-Resist we will uncover novel physical and genetic interactions, identify coordination mechanisms and elucidate their impact on bacterial physiology and antibiotic resistance. To this end, we will focus on three aims: (i) Explore the assembly of lipopolysaccharide (LPS), an endotoxin covering the bacterial surface, and characterize its interplay with envelope biogenesis. (ii) Comprehensively map PG-OMPs interactions and study how they affect bacterial response to antibiotics. (iii) Uncover the outcomes of spatially heterogenous envelope biogenesis and whether this process is a source of phenotypic diversity. To address these questions, a multidisciplinary approach will be taken, combining cutting-edge microscopy, advanced genetic screens, proteomics and novel functionality assays. We expect this project to uncover the interaction network amongst envelope components, reveal how it impacts the physiology of both single cells and bacterial populations, and ultimately expose the secrets of bacterial resilience.

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(opens in new window) ERC-2024-STG

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Host institution

THE HEBREW UNIVERSITY OF JERUSALEM
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 499 894,00
Address
EDMOND J SAFRA CAMPUS GIVAT RAM
91904 JERUSALEM
Israel

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Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 499 894,00

Beneficiaries (1)

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