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A nanobody imaging tool to detect protein crystals and predict therapeutic response in asthmatics

Project description

Innovative imaging for precision asthma therapy

Asthma is a chronic respiratory condition characterised by inflammation and narrowing of the airways, leading to symptoms like shortness of breath. A key cause is the abnormal production of thick, sticky mucus which accumulates in the airways and obstructs normal airflow. Eosinophils play a role in asthma, as they contribute to mucus stickiness and hinder its clearance. The ERC-funded CRYSTAL-SHINE project has developed crystal-dissolving antibodies that improve mucus clearance in mice. However, further clinical development is hindered by the lack of a biomarker for mucus that is rich in Charcot-Leyden crystals (CLCs). To overcome this obstacle, CRYSTAL-SHINE will develop a PET tracer for non-invasive imaging of CLCs, thereby improving the monitoring of antibody treatment.

Objective

Asthma is characterized by airway obstruction, causing difficulty in breathing. The major cause of therapy resistant obstruction is mucus plug buildup in 60% of severe asthmatics. Mucus plugs form as a result of abnormal mucus production by airway barrier cells and recruitment of eosinophils that change the mucus from a gel- to a glue-like state. Eosinophils also release galectin-10 (Gal10) protein, which crystallizes and forms Charcot-Leyden crystals (CLC). My advanced ERC grant unraveled how CLCs damage the epithelium to aggravate airway inflammation, cause more mucus build-up and physically impede the clearance of plugs, because of their needle-like shape. We therefore developed crystal-dissolving antibodies, that improve mucus clearance in mice. Meanwhile we also found that chronic rhinosinusitis (CRS) and 10% of cystic fibrosis (CF) patients have CLC-rich mucus. The current problem we are facing in the further clinical development of crystal dissolving antibodies is that we do not have a biomarker for CLC-rich mucus, and therefore it would be hard to predict which patients benefit from treatment with CLC-dissolving antibodies. The solution is to develop a non-invasive imaging technique to visualize crystal-containing mucus. We have therefore generated a Gal10 PET tracer. We have
developed humanized mice that express Gal10 and form CLCs in their airways, and will be used to test the feasibility of Gal10 PET tracer imaging. A major challenge is to find the best conjugation format of the PET
biomarker, and unravel the pharmacokinetics and - dynamics of the imaging modality. In parallel, we will test if Gal10 antibodies bind to human biopsies from asthma, CRS and CF patients, and how this biomarker correlates to CLC presence. Gaining PoC that Gal10 PET imaging works as a biomarker to detect CLCs will personalize the approach and create deal-making value for our antibody program.

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HORIZON-ERC-POC - HORIZON ERC Proof of Concept Grants

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Call for proposal

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(opens in new window) ERC-2024-POC

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Host institution

VIB VZW
Net EU contribution

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€ 150 000,00
Address
SUZANNE TASSIERSTRAAT 1
9052 ZWIJNAARDE - GENT
Belgium

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Region
Vlaams Gewest Prov. Oost-Vlaanderen Arr. Gent
Activity type
Research Organisations
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Total cost

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Beneficiaries (1)

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