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Understanding the mechanisms of neuronal secretory autophagy

Project description

Uncovering neuronal secretory pathways

Neurons experience metabolic stress causing damage to proteins and organelles, which then must be cleared to maintain function. While autophagy degrades most of this cellular debris, neurons also secrete some of it into the extracellular space (a secretory pathway), a process that is still poorly understood. Dysregulation of this process is linked to neurodegenerative diseases such as Alzheimer’s. Supported by the Marie Skłodowska-Curie Actions programme, the NEURO-SECRET project aims to use human induced pluripotent stem cell-derived neurons and state-of-the-art molecular tools to visualise neuronal secretory autophagy at high spatiotemporal resolution. By combining bioluminescent/fluorescent reporters with proximity labelling and chemogenetic relocalisation, the project will chart critical regulators and explore ways to modulate this pathway, shedding new light on probing and even treating neurodegeneration.

Objective

Neurons transmit information by connecting with other neurons over long distances through their dendrites and axons. Repeated stimulation places a high metabolic demand on these cells, leading to the accumulation of defective organelles and protein aggregates. To maintain protein homeostasis, neurons rely on constitutive autophagy to remove these damaged proteins and organelles from their dendrites and axons, targeting them for degradation. However, it is becoming increasingly clear that neurons also secrete a part of this autophagic cargo into the extracellular space. The mechanisms governing neuronal secretory autophagy are still poorly understood. The factors that determine whether autophagic cargo is degraded or secreted, as well as the precise neuronal location from which secretory autophagy occurs, remain unknown. Understanding these mechanisms is highly relevant in neurodegenerative diseases, such as Alzheimers disease, where the balance between degradative and secretory autophagy is disrupted. In this interdisciplinary project, I will use human iPSC-derived neurons and develop novel molecular tools to elucidate the mechanisms underlying neuronal secretory autophagy. I will employ state-of-the-art bioluminescent and fluorescent reporters to characterize this process with high spatiotemporal resolution, leveraging my experience in studying exosome release during my PhD. Furthermore, I will apply the host labs expertise in proximity labeling and chemogenetic relocalization to identify key regulators of neuronal secretory autophagy and explore the potential of modulating secretory autophagy in a cell model for Alzheimers disease. Ultimately, this project aims to provide fundamental insights into the mechanisms driving neuronal secretory autophagy and the potential for modulating this process in neurodegenerative diseases.

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HORIZON-TMA-MSCA-PF-EF - HORIZON TMA MSCA Postdoctoral Fellowships - European Fellowships

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Call for proposal

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(opens in new window) HORIZON-MSCA-2024-PF-01

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Coordinator

UNIVERSITEIT UTRECHT
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 232 916,16
Address
HEIDELBERGLAAN 8
3584 CS Utrecht
Netherlands

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Activity type
Higher or Secondary Education Establishments
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Total cost

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