Skip to main content
Aller à la page d’accueil de la Commission européenne (s’ouvre dans une nouvelle fenêtre)
français français
CORDIS - Résultats de la recherche de l’UE
CORDIS
CORDIS Web 30th anniversary CORDIS Web 30th anniversary

Novel Matrix Stiffness-regulated Genes in Lymphangiogenesis and Angiogenesis

Description du projet

Et au milieu coule une rivière: formation des vaisseaux sanguins et lymphatiques dans la matrice extracellulaire

Le sang et la lymphe circulent dans le corps dans des vaisseaux sanguins et lymphatiques, respectivement, qui forment des réseaux complexes à travers les tissus. Le sang transporte les gaz, les nutriments et les déchets métaboliques. Le système lymphatique draine les liquides et les protéines excédentaires des tissus de l’organisme et les renvoie dans la circulation sanguine, prévenant ainsi les gonflements. Des chercheurs ont récemment montré que la rigidité des matrices extracellulaires (MEC) affecte la formation de nouveaux vaisseaux lymphatiques. Avec le soutien du programme Actions Marie Skłodowska-Curie, le projet STRomA étudie le rôle de la rigidité de la MEC dans la formation de nouveaux capillaires et vaisseaux lymphatiques, dans le but de mettre en évidence les similitudes et les différences mécanistiques.

Objectif

Endothelial cells (ECs) recognize and respond to mechanical forces through their cell-cell and cell-matrix adhesions and translate physical stimuli into biological responses in a process called mechanotransduction. The composition and mechanical properties of the extracellular matrix (ECM) differ across the vascular tree, in its surrounding tissues and in development and diseases, such as edema formation. I have recently shown for the first time that ECM stiffness fundamentally controls lymphangiogenesis. I hypothesize that changes in ECM stiffness are a key regulatory mechanism of angiogenic processes in development and disease. A comprehensive analysis of novel ECM stiffness-regulated genes is pivotal to understand these processes integrally. In a preliminary study, I have performed differential RNA sequencing of blood (B) and lymphatic (L) ECs cultured on soft and stiff matrices. 3200 genes were regulated similarly in BECs and LECs in response to changes in matrix stiffness. Interestingly, the same number of genes was differently regulated. In the next two years, I will study the role of selected genes in lymphangiogenesis and angiogenesis in vitro and in transgenic mouse models with state-of-the-art microscope and live imaging techniques. First, I will analyze an actin-regulating protein family that is significantly regulated by matrix stiffness in both, BEC and LECs, suggesting a more general role in lymphangiogenesis and angiogenesis by regulating cytoskeletal dynamics. Second, I will study a molecule, which is involved in intracellular cGMP signaling and is predominantly regulated in LECs, suggesting a more specific role in lymphangiogenesis. Last, I will generate an in vitro fluorescent stiffness sensor to live-visualize changes in stiffness inside the EC. Ultimately, the proposed action can provide novel targets to modulate lymph and blood vessel formation with implications for edema treatment and will support me to become an independent group leader.

Champ scientifique (EuroSciVoc)

CORDIS classe les projets avec EuroSciVoc, une taxonomie multilingue des domaines scientifiques, grâce à un processus semi-automatique basé sur des techniques TLN. Voir: https://op.europa.eu/en/web/eu-vocabularies/euroscivoc.

Vous devez vous identifier ou vous inscrire pour utiliser cette fonction

Régime de financement

MSCA-IF-EF-ST - Standard EF

Coordinateur

UNIVERSITAETSKLINIKUM HAMBURG-EPPENDORF
Contribution nette de l'UE
€ 162 806,40
Coût total
€ 162 806,40