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The role of degradation pathways on cell stemness and fate determination

Descripción del proyecto

El papel de las vías de degradación en el fenotipo de las células madre

Las células madre hematopoyéticas (CMH) tienen la capacidad de autorrenovarse y diferenciarse en cualquier glóbulo sanguíneo maduro. Mediante la división celular asimétrica y una compleja red de eventos de regulación a nivel transcripcional, traslacional, epigenético y metabólico, estas células madre dan lugar a células de destinos diferentes. El proyecto Asymmetric fates, financiado con fondos europeos, investigará el papel de la autofagia y de la degradación del proteasoma en la determinación del destino celular en el sistema hematopoyético. Los científicos descifrarán, mediante imágenes de la herencia asimétrica de diferentes componentes en CMH, los efectos de las vías de degradación en el destino celular. Se espera que los conocimientos generados hagan avanzar las aplicaciones de las CMH en medicina regenerativa.

Objetivo

The haematopoietic system relies on the potential of haematopoietic stem cells (HSCs) to self-renew and differentiate into all lineages of mature blood cells, and is a reference model to study differentiation hierarchies. Cell fate determination results from different layers of regulation, including transcriptional, translational, epigenetic, metabolic, and cell biological changes. Degradation pathways, such as autophagy and the proteasome, play mechanistically relevant roles that in principle may impact on all these layers. Indeed, catabolic degradation results in the building blocks necessary for anabolic processes, while it also preserves stemness and regenerative potential. Asymmetric cell division (ACD) has been extensively reported to contribute to maintenance of stemness by the rise of daughters with divergent fates in stem cells, including HSCs. Taken together, I postulate that degradation pathways work synergistically to give rise to the asymmetric fates observed in HSCs differentiation and can be targeted for future therapeutic use in humans. I plan to test this by 1. establishing an efficient strategy to image asymmetric inheritance of cargo by long-term ex vivo HSCs expansion, 2. verifying whether autophagosomes and proteasomes are co-inherited in HSCs mitoses, and 3. assessing the impact of cargo segregation by ACD on HSC maintenance and differentiation. I will use state-of-the-art techniques and novel murine models to assess the molecular and cell biological mechanisms of ACD modulation on HSC maintenance, relying on imaging of known and potentially novel components that are asymmetrically inherited by HSCs and able to impact their fate determination. Finally, I will further evaluate the in vivo impact of cargo inheritance on haematopoiesis by using single-cell transplantation. The knowledge derived from this project will potentially boost the development of novel regenerative medicine therapeutic approaches.

Ámbito científico (EuroSciVoc)

CORDIS clasifica los proyectos con EuroSciVoc, una taxonomía plurilingüe de ámbitos científicos, mediante un proceso semiautomático basado en técnicas de procesamiento del lenguaje natural.

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Coordinador

THE CHANCELLOR, MASTERS AND SCHOLARS OF THE UNIVERSITY OF OXFORD
Aportación neta de la UEn
€ 224 933,76
Dirección
WELLINGTON SQUARE UNIVERSITY OFFICES
OX1 2JD Oxford
Reino Unido

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Región
South East (England) Berkshire, Buckinghamshire and Oxfordshire Oxfordshire
Tipo de actividad
Higher or Secondary Education Establishments
Enlaces
Coste total
€ 224 933,76