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Applying human lung tissue models to develop a personalised treatment for critically ill

Descrizione del progetto

Una terapia priva di cellule per la sindrome da distress respiratorio acuto

La sindrome da distress respiratorio acuto è una grave patologia infiammatoria dei polmoni che insorge nei pazienti in condizioni critiche. Nonostante per il suo trattamento stiano emergendo come promettente strumento terapeutico le cellule staminali mesenchimali, il rischio di formazione tumorale ne impedisce l’utilizzo diffuso. Il progetto ALGORITM, finanziato dall’UE, si propone di sviluppare e testare una terapia priva di cellule per la sindrome da distress respiratorio, basata sulle vescicole extracellulari secrete dalle cellule staminali mesenchimali. Avvalendosi di modelli di tessuto polmonare umano, gli scienziati del progetto valuteranno il potenziale terapeutico di queste vescicole e le correlate variazioni tessutali e fornirà prove delle implicazioni trascrittomiche, metaboliche e funzionali della loro somministrazione, aprendo la strada all’impiego efficace in ambito clinico di questo approccio.

Obiettivo

Acute respiratory distress syndrome (ARDS) is a major cause of mortality in the critically ill patients with no effective pharmacological treatment. Recently, two distinct endotypes of ARDS were identified and validated in four large randomized controlled trials: a hyper-inflammatory and a hypo-inflammatory endotype. These endotypes have strikingly different clinical characteristics, biomarker profiles, clinical outcomes, and treatment responses. While these data are highly promising, the biology of these endotypes is not understood. Mesenchymal Stem Cells (MSCs) hold significant therapeutic promise for ARDS and are rapidly progressing to clinical trials across the world. However, concern for the use of stem cells, specifically the risk of tumor formation, remains unresolved. Accumulating evidence suggests that MSCs exert their therapeutic benefit through the release of extracellular vesicles (EVs). MSC EVs are being actively developed as a cell free therapy for ARDS, but for the effective clinical translation it is important to establish EV therapeutic potential for both biological endotypes of patients. In this project, I am going to use cutting edge technology and employ the most physiological human lung tissue models to date to study the biology of ARDS endotypes and to test therapeutic potential of MSC EVs. The main objectives are: i) To reveal how endotype–specific environments alter human lung tissue and how these changes are affected by EV treatment at a single cell level, using single cell RNA-Seq transcriptomic profiling of precision-cut human lung tissue slices, ii) to study how endotype-specific environments (with or without EV treatment) alter functional properties of the alveolar epithelial-endothelial barrier using lung-on-a-chip microfluidic model, iii) to study how endotype-specific environments (with or without EVs) alter metabolism of the primary human pulmonary cells involved in ARDS pathophysiology using Seahorse Metabolic Analyser.

Campo scientifico (EuroSciVoc)

CORDIS classifica i progetti con EuroSciVoc, una tassonomia multilingue dei campi scientifici, attraverso un processo semi-automatico basato su tecniche NLP. Cfr.: https://op.europa.eu/en/web/eu-vocabularies/euroscivoc.

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Coordinatore

THE QUEEN'S UNIVERSITY OF BELFAST
Contributo netto dell'UE
€ 212 933,76
Indirizzo
UNIVERSITY ROAD LANYON BUILDING
BT7 1NN Belfast
Regno Unito

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Regione
Northern Ireland Northern Ireland Belfast
Tipo di attività
Higher or Secondary Education Establishments
Collegamenti
Costo totale
€ 212 933,76