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Mechano-Regulation of Proteins at Low Forces: Paving the Way for Therapeutic Interventions

Project description

The role of mechanical forces in protein activity

Many biological processes including cell division, cell migration and development involve the sensing and transduction of mechanical forces. Proteins are an integral part of these processes, undergoing conformational changes in response to such forces. The EU-funded ProForce project aims to develop a method for measuring the impact of mechanical forces on single proteins. Researchers will focus on proteins known to be activated by force stimuli, such as the haemostatic von Willebrand factor, the focal adhesion kinase, which is implicated in cell migration and metastasis, and G-protein coupled receptors. Results will offer important insight into the ability of mechanical forces to regulate protein activity and may disclose novel targets for therapeutic intervention.

Objective

Mechanical forces play critical roles in the regulation of biological functions, including development, motility, and haemostasis. Aberrant mechano-regulation is implicated in human pathologies, including cancer and infarction. Proteins sense forces by undergoing conformational changes under external loads that trigger downstream signaling. Despite its importance, mechanical regulation at the single-protein level remains poorly understood, in part due to a lack of suitable techniques to probe the physiological highly relevant low force (~1 pN) range. ProForce aims to understand mechano-regulation at the single-molecule level in this previously inaccessible regime and to develop approaches to directly interfere with and correct aberrant force responses. We propose to advance massively-parallel magnetic tweezers as the ideal tool for single-protein force measurements, as they can resolve very small forces (<0.1 pN), perform stable and highly-multiplexed measurements over long times, and can readily be combined with fluorescence detection to provide an orthogonal read out. We aim to address three sets of biological systems that are regulated by low forces and are potential targets for drugs that alter their force response: 1) The blood protein von Willebrand factor (VWF) that is activated by shear flow and critically involved in haemostasis. Reduced VWF activity leads to bleeding disorders, while gain-of-function mutations increase the risk of myocardial infarction. 2) Focal adhesion kinase is a force-activated kinase involved in intracellular signal transduction pathways important in cell proliferation, migration, and metastasis. 3) Adhesion G-protein coupled receptors are a subset of the GPCR-family, activated by mechanical stimuli and involved in development, immunity, neuronal function, and tumorigenesis. The aim of ProForce is to understand mechano-regulation at the single-protein level and to establish force response as a potential drug target.

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Topic(s)

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ERC-COG - Consolidator Grant

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Call for proposal

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(opens in new window) ERC-2020-COG

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Host institution

UNIVERSITAET AUGSBURG
Net EU contribution

Net EU financial contribution. The sum of money that the participant receives, deducted by the EU contribution to its linked third party. It considers the distribution of the EU financial contribution between direct beneficiaries of the project and other types of participants, like third-party participants.

€ 1 049 351,00
Address
UNIVERSITAETSSTRASSE 2
86159 Augsburg
Germany

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Region
Bayern Schwaben Augsburg, Kreisfreie Stadt
Activity type
Higher or Secondary Education Establishments
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Total cost

The total costs incurred by this organisation to participate in the project, including direct and indirect costs. This amount is a subset of the overall project budget.

€ 1 049 351,00

Beneficiaries (2)

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