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Microbial genome defence pathways: from molecular mechanisms to next-generation molecular tools

Projektbeschreibung

Molekulare Einblicke in mikrobielle Abwehrmechanismen

Von Restriktionsenzymen bis hin zu CRISPR-Cas-Nuklease-basierten Editiersystemen haben Prokaryoten eine ganze Reihe von Immunreaktionen zur Bekämpfung von Parasiten entwickelt. Gleichzeitig haben sich diese Abwehrsysteme in den Händen der Wissenschaft als wertvolle Werkzeuge für die Gentechnik und die Genomchirurgie erwiesen. In den vergangenen Jahren wurden die Vor- und Nachteile der CRISPR-Geneditierungstechnologie deutlich, was die Notwendigkeit einer weiteren Entwicklung unterstreicht. Vor diesem Hintergrund wird das EU-finanzierte Projekt CRISPR2.0 die molekularen Strukturen und Mechanismen alternativer CRISPR-basierter Systeme untersuchen. Letztlich geht es darum, die Evolution der Abwehrmechanismen des Genoms zu verstehen und neuartige Genomchirurgie-Werkzeuge zu erstellen.

Ziel

The constant arms race between prokaryotic microbes and their molecular parasites such as viruses, plasmids and transposons has driven the evolution of complex genome defence mechanisms. The CRISPR-Cas defence systems provide adaptive RNA-guided immunity against invasive nucleic acid elements. CRISPR-associated effector nucleases such as Cas9, Cas12a and Cas13 have emerged as powerful tools for precision genome editing, gene expression control and nucleic acid detection. However, these technologies suffer from drawbacks that limit their efficacy and versatility, necessitating the search for additional exploitable molecular activities. Building on our recent structural and biochemical studies, the goal of this project is to investigate the molecular architectures and mechanisms of CRISPR-associated systems and other genome defence mechanisms, aiming not only to shed light on their biological roles but also inform their technological development. Specifically, the proposed studies will examine (i) the molecular basis of cyclic oligoadenylate signalling in type III CRISPR-Cas systems, (ii) the mechanism of transposon-associated type I CRISPR-Cas systems and their putative function in RNA-guided DNA transposition, and (iii) molecular activities associated with recently described non-CRISPR defence systems. Collectively, the proposed studies will advance our understanding of the molecular functions of genome defence mechanisms in shaping the evolution of prokaryotic genomes and make critical contributions to their development as novel genetic engineering tools.

Gastgebende Einrichtung

UNIVERSITAT ZURICH
Netto-EU-Beitrag
€ 1 996 525,00
Adresse
RAMISTRASSE 71
8006 Zurich
Schweiz

Auf der Karte ansehen

Region
Schweiz/Suisse/Svizzera Zürich Zürich
Aktivitätstyp
Higher or Secondary Education Establishments
Links
Gesamtkosten
€ 1 996 525,00

Begünstigte (1)